Correction| Volume 193, ISSUE 5, P654, May 2023

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        The authors of the article entitled, “Gonadotropin-Releasing Hormone Stimulates Biliary Proliferation by Paracrine/Autocrine Mechanisms” (Volume 185, pages 1061–1072 of the April 2015 issue of The American Journal of Pathology; DOI: have discovered an error in Figure 3B and are replacing the panels with the images provided below. Previously published staining in the normal rat liver GnRH image had post-editing issues leading to areas of overlapping hepatocyte nuclei. While these images were not quantified and the post-editing did not alter the published analysis, the authors have chosen to replace both the normal and bile duct–ligated rat liver images stained for GnRH to maintain consistency of the staining. The updated images reflect the replaced staining for GnRH in both normal and bile duct–ligated rat liver sections. The original legend for this figure is correct as published.

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        • Gonadotropin-Releasing Hormone Stimulates Biliary Proliferation by Paracrine/Autocrine Mechanisms
          The American Journal of PathologyVol. 185Issue 4
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            During cholestatic liver disease, there is dysregulation in the balance between biliary growth and loss in bile duct–ligated (BDL) rats modulated by neuroendocrine peptides via autocrine/paracrine pathways. Gonadotropin-releasing hormone (GnRH) is a trophic peptide hormone that modulates reproductive function and proliferation in many cell types. We evaluated the autocrine role of GnRH in the regulation of cholangiocyte proliferation. The expression of GnRH receptors was assessed in a normal mouse cholangiocyte cell line (NMC), sham, and BDL rats.
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